Dating a cancer female
E6 also has a close relationship with the cellular protein E6-associated protein (E6-AP), which is involved in the ubiquitin ligase pathway, a system that acts to degrade proteins.E6-AP binds ubiquitin to the p53 protein, thereby flagging it for proteosomal degradation.Genital warts are quite contagious, while common, flat, and plantar warts are much less likely to spread from person to person.HPV infection of the skin in the genital area is the most common sexually transmitted infection worldwide.Immunological control is HPV type-specific, meaning an individual may become resistant to one HPV type while remaining susceptible to other types.In one study, infection by HPV types 2, 27, and 57 was found in people with warts, while infection by HPV types 1, 2, 63, and 27 was found in people with clinically normal skin.Moreover, findings indicate this type of cancer is much more prevalent in men than in women, something that needs to be further explored.
Because the process of transforming normal cervical cells into cancerous ones is slow, cancer occurs in people having been infected with HPV for a long time, usually over a decade or more (persistent infection).In the United States, HPV is expected to replace tobacco as the main causal agent for oral cancer, and the number of newly diagnosed, HPV-associated head and neck cancers is expected to surpass that of cervical cancer cases by 2020.In recent years, the United States has experienced an increase in the number of cases of throat cancer caused by HPV type 16.All HPVs are believed to be capable of establishing long-term "latent" infections in small numbers of stem cells present in the skin.Although these latent infections may never be fully eradicated, immunological control is thought to block the appearance of symptoms such as warts.
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Some HPV types, such as HPV-5, may establish infections that persist for the lifetime of the individual without ever manifesting any clinical symptoms.